Polymorphisms of STK15 (Aurora-A) gene and lung cancer risk in Caucasians

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15 صفحه اول

STK15 polymorphisms and association with risk of invasive ovarian cancer.

STK15 is a putative oncogene that codes for a centrosome-associated, serine/threonine kinase, the normal function of which is to ensure accurate segregation of chromosomes during mitosis. Amplification of STK15 has been reported in ovarian tumors, suggesting a role in ovarian cancer pathology. STK15 is polymorphic with two single nucleotide substitutions (449t/a and 527g/a) in evolutionarily co...

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Synergistic effects of STK15 gene polymorphisms and endogenous estrogen exposure in the risk of breast cancer.

STK15 is a member of a family of serine/threonine kinases that act as key regulators of chromosome segregation and cytokinesis. Over expression of the STK15 gene leads to centrosome amplification, chromosomal instability, aneuploidy, and transformation. It has been reported that the 91T --> A (Phe --> Ile at codon 31) polymorphism in the STK15 gene affects the function of this gene. We hypothes...

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FokI and BsmI Polymorphisms of the VDR Gene and Breast Cancer Risk

Introduction: Vitamin D fulfills its crucial role in cell proliferation and death through signal transduction into the nucleus by vitamin D receptor (VDR). Recent studies have depicted the association between VDR gene polymorphisms and different cancers, including breast cancer. This study attempted to consider the relationship between VDR gene polymorphisms and breast cancer risk among women i...

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FokI and BsmI Polymorphisms of the VDR Gene and Breast Cancer Risk

Introduction: Vitamin D fulfills its crucial role in cell proliferation and death through signal transduction into the nucleus by vitamin D receptor (VDR). Recent studies have depicted the association between VDR gene polymorphisms and different cancers, including breast cancer. This study attempted to consider the relationship between VDR gene polymorphisms and breast cancer risk among women i...

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ژورنال

عنوان ژورنال: Carcinogenesis

سال: 2006

ISSN: 0143-3334,1460-2180

DOI: 10.1093/carcin/bgl149